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Title | Published in | Access level | OA Policy | Year | Views | Downloads | |
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Cell-type specific synaptic plasticity in dorsal striatum is associated with punishment-resistance compulsive-like cocaine self-administration in mice | Neuropsychopharmacology | 2023 | 149 | 59 | |||
Synaptic mechanism underlying serotonin modulation of transition to cocaine addiction | Science | 2021 | 310 | 148 | |||
Corticostriatal activity driving compulsive reward seeking | Biological psychiatry | 2021 | 249 | 204 | |||
Regulation of GluA1 phosphorylation by d-amphetamine and methylphenidate in the cerebellum | Addiction Biology | 2020 | 193 | 0 | |||
Projection-specific deficits in synaptic transmission in adult Sapap3-knockout mice | Neuropsychopharmacology | 2020 | 251 | 94 | |||
Social transmission of food safety depends on synaptic plasticity in the prefrontal cortex | Science | 2019 | 367 | 6 | |||
Dopamine neurons projecting to medial shell of the nucleus accumbens drive heroin reinforcement | eLife | 2018 | 405 | 228 | |||
Stochastic synaptic plasticity underlying compulsion in a model of addiction | Nature | 2018 | 658 | 664 | |||
Cell-Type Specific Insertion of GluA2-Lacking AMPARs with Cocaine Exposure Leading to Sensitization, Cue-Induced Seeking, and Incubation of Craving | Neuropsychopharmacology | 2016 | 601 | 27 | |||
[Mesolimbic dopamine neuron stimulation is sufficient for the progression to addiction] | MS. Médecine sciences | 2016 | 460 | 0 | |||
Addiction therapy. Refining deep brain stimulation to emulate optogenetic treatment of synaptic pathology | Science | 2015 | 735 | 118 | |||
Sufficiency of Mesolimbic Dopamine Neuron Stimulation for the Progression to Addiction | Neuron | 2015 | 708 | 98 | |||
Accumbal D1R Neurons Projecting to Lateral Hypothalamus Authorize Feeding | Neuron | 2015 | 570 | 3 | |||
Optogenetic dissection of neural circuitry: from synaptic causalities to blue prints for novel treatments of behavioral diseases | Current opinion in neurobiology | 2015 | 583 | 2 | |||
Contrasting forms of cocaine-evoked plasticity control components of relapse | Nature | 2014 | 1,035 | 802 | |||
D1R/GluN1 complexes in the striatum integrate dopamine and glutamate signalling to control synaptic plasticity and cocaine-induced responses | Molecular psychiatry | 2014 | 579 | 1 | |||
Cocaine disinhibits dopamine neurons by potentiation of GABA transmission in the ventral tegmental area | Science | 2013 | 703 | 413 | |||
Approche optogénétique de suppression de la plasticité synaptique induite par la cocaïne | MS. Médecine sciences | 2012 | 617 | 1 | |||
Reversal of cocaine-evoked synaptic potentiation resets drug-induced adaptive behaviour | Nature | 2012 | 959 | 693 | |||
A comparison of striatal-dependent behaviors in wild-type and hemizygous Drd1a and Drd2 BAC transgenic mice | The Journal of neuroscience | 2012 | 549 | 0 |