| Acceleration of human myoblast fusion by depolarization: graded Ca2+ signals involved | Development |  | | 2003 | 619 | 0 |
|
| Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels | The Journal of cell biology | | | 2001 | 604 | 0 |
|
| T-type alpha 1H Ca2+ channels are involved in Ca2+ signaling during terminal differentiation (fusion) of human myoblasts | Proceedings of the National Academy of Sciences of the United States of America | | | 2000 | 633 | 0 |
|
| Mibefradil (Ro 40-5967) inhibits several Ca2+ and K+ currents in human fusion-competent myoblasts | British journal of pharmacology | | | 1999 | 548 | 0 |
|
| An ether -a-go-go K+ current, Ih-eag, contributes to the hyperpolarization of human fusion-competent myoblasts | Journal of physiology | | | 1998 | 653 | 0 |
|
| Role of an inward rectifier K+ current and of hyperpolarization in human myoblast fusion | Journal of physiology | | | 1998 | 612 | 0 |
|
| Cloning of a human ether-a-go-go potassium channel expressed in myoblasts at the onset of fusion | FEBS letters | | | 1998 | 589 | 0 |
|
| Role of nicotinic acetylcholine receptors at the vertebrate myotendinous junction: a hypothesis | Neuromuscular disorders | | | 1996 | 613 | 0 |
|
| Contribution of a non-inactivating potassium current to the resting membrane potential of fusion-competent human myoblasts | Journal of physiology | | | 1996 | 711 | 0 |
|
