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Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23

Published inPloS one, vol. 12, no. 6, e0180035
Publication date2017
Research group
Citation (ISO format)
MADON-SIMON, Marta et al. Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23. In: PloS one, 2017, vol. 12, n° 6, p. e0180035. doi: 10.1371/journal.pone.0180035
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ISSN of the journal1932-6203
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Creation06/26/2017 9:37:00 PM
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Update time03/15/2023 1:48:22 AM
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