Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23
ContributorsMadon-Simon, Marta; Grad, Iwona; Bayo, Pilar; Pérez, Paloma; Picard, Didier
Published inPloS one, vol. 12, no. 6, e0180035
Publication date2017
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MADON-SIMON, Marta et al. Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23. In: PloS one, 2017, vol. 12, n° 6, p. e0180035. doi: 10.1371/journal.pone.0180035
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- PID : unige:95099
- DOI : 10.1371/journal.pone.0180035
Additional URL for this publicationhttp://dx.plos.org/10.1371/journal.pone.0180035
Journal ISSN1932-6203