Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23

Madon-Simon, Marta; Grad, Iwona; Bayo, Pilar; Pérez, Paloma; Picard, Didier

doi:10.1371/journal.pone.0180035

Scientific article

English

Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23

ContributorsMadon-Simon, Marta; Grad, Iwona; Bayo, Pilar; Pérez, Paloma; Picard, Didier

Published inPloS one, vol. 12, no. 6, e0180035

Publication date2017

Affiliation entities

Faculté des sciences / Section de biologie / Département de biologie cellulaire

Research groups

Groupe Picard

Citation (ISO format)

MADON-SIMON, Marta et al. Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23. In: PloS one, 2017, vol. 12, n° 6, p. e0180035. doi: 10.1371/journal.pone.0180035