Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23

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Title		Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23
Authors		Madon-Simon, Marta Grad, Iwona Bayo, Pilar Pérez, Paloma Picard, Didier
Published in		PLOS ONE. 2017, vol. 12, no. 6, p. e0180035
Identifiers		DOI: 10.1371/journal.pone.0180035
Full text		Article (Published version) (17.3 MB) - Free access Other version: http://dx.plos.org/10.1371/journal.pone.0180035
Structures		Faculté des sciences / Section de biologie / Département de biologie cellulaire
Research group		Groupe Picard
Citation (ISO format)		MADON-SIMON, Marta et al. Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23. In: PLOS ONE, 2017, vol. 12, n° 6, p. e0180035. https://archive-ouverte.unige.ch/unige:95099