Scientific article
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Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23

Published inPloS one, vol. 12, no. 6, e0180035
Publication date2017
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Citation (ISO format)
MADON-SIMON, Marta et al. Defective glucocorticoid receptor signaling and keratinocyte-autonomous defects contribute to skin phenotype of mouse embryos lacking the Hsp90 co-chaperone p23. In: PloS one, 2017, vol. 12, n° 6, p. e0180035. doi: 10.1371/journal.pone.0180035
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Article (Published version)
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Additional URL for this publicationhttp://dx.plos.org/10.1371/journal.pone.0180035
Journal ISSN1932-6203
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Creation26/06/2017 21:37:00
First validation26/06/2017 21:37:00
Update time15/03/2023 01:48:22
Status update15/03/2023 01:48:22
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