MHC Class II-Restricted Antigen Presentation by Plasmacytoid Dendritic Cells Drives Proatherogenic T Cell Immunity

Sage, Andrew P; Murphy, Deirdre; Maffia, Pasquale; Masters, Leanne M; Sabir, Suleman R; Baker, Lauren L; Cambrook, Helen; Finigan, Alison J; Ait-Oufella, Hafid; Grassia, Gianluca; Harrison, James E; Ludewig, Burkhard; Reith, Walter; Hansson, Göran K; Reizis, Boris; Hugues, Stéphanie; Mallat, Ziad

doi:10.1161/CIRCULATIONAHA.114.011090

Scientific article

English

MHC Class II-Restricted Antigen Presentation by Plasmacytoid Dendritic Cells Drives Proatherogenic T Cell Immunity

ContributorsSage, Andrew P; Murphy, Deirdre; Maffia, Pasquale; Masters, Leanne M; Sabir, Suleman R; Baker, Lauren L; Cambrook, Helen; Finigan, Alison J; Ait-Oufella, Hafid; Grassia, Gianluca; Harrison, James E; Ludewig, Burkhard; Reith, Walter; Hansson, Göran K; Reizis, Boris; Hugues, Stéphanie; Mallat, Ziad

Published inCirculation, vol. 130, no. 16, p. 1363-1373

Publication date2014

Abstract

Plasmacytoid dendritic cells (pDCs) bridge innate and adaptive immune responses and are important regulators of immuno-inflammatory diseases. However, their role in atherosclerosis remains elusive.

Affiliation entities

Faculté de médecine / Section de médecine fondamentale / Département de pathologie et immunologie

Research groups

Groupe Reith Walter (pathologie et immunologie) (282)

Citation (ISO format)

SAGE, Andrew P et al. MHC Class II-Restricted Antigen Presentation by Plasmacytoid Dendritic Cells Drives Proatherogenic T Cell Immunity. In: Circulation, 2014, vol. 130, n° 16, p. 1363–1373. doi: 10.1161/CIRCULATIONAHA.114.011090

Article (Published version)

Identifiers

PID : unige:44167
DOI : 10.1161/CIRCULATIONAHA.114.011090
PMID : 25223984

Journal ISSN0009-7322

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Creation25/11/2014 14:52:00

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MHC Class II-Restricted Antigen Presentation by Plasmacytoid Dendritic Cells Drives Proatherogenic T Cell Immunity

Plasmacytoid dendritic cells (pDCs) bridge innate and adaptive immune responses and are important regulators of immuno-inflammatory diseases. However, their role in atherosclerosis remains elusive.

Technical informations