Increased expression of adenosine triphosphate-sensitive K+ channels in mitral dysfunction: mechanically stimulated transcription and hypoxia-induced protein stability?
Published inJournal of the American College of Cardiology, vol. 59, no. 4, p. 390-396
Publication date2012
Abstract
Keywords
- Aged
- Animals
- Anoxia/metabolism
- Blood Gas Analysis
- Blood Pressure
- Echocardiography
- Female
- Gene Expression Regulation
- Heart Ventricles/metabolism
- Humans
- Hypoxia-Inducible Factor 1
- Alpha Subunit/metabolism
- KATP Channels/metabolism
- Male
- Mice
- Knockout
- Middle Aged
- Mitral Valve Insufficiency/metabolism
- Myocardium/metabolism
- Oxygen/physiology
- Partial Pressure
- Potassium Channels
- Inwardly Rectifying/metabolism
- Protein Stability
- RNA
- Messenger/metabolism
- Stress
- Mechanical
Research groups
Citation (ISO format)
RAEIS, Véronique et al. Increased expression of adenosine triphosphate-sensitive K+ channels in mitral dysfunction: mechanically stimulated transcription and hypoxia-induced protein stability? In: Journal of the American College of Cardiology, 2012, vol. 59, n° 4, p. 390–396. doi: 10.1016/j.jacc.2011.08.077
Main files (1)
Article (Published version)
Identifiers
- PID : unige:26258
- DOI : 10.1016/j.jacc.2011.08.077
- PMID : 22133355
Journal ISSN0735-1097