G9a dictates neuronal vulnerability to inflammatory stress via transcriptional control of ferroptosis
Publié dansScience advances, vol. 8, no. 31, eabm5500
Date de publication2022-08-05
Date de mise en ligne2022-08-05
Résumé
Mots-clés
- Animals
- Encephalomyelitis, Autoimmune, Experimental / genetics
- Ferroptosis / genetics
- Gene Expression Regulation
- Histone-Lysine N-Methyltransferase / metabolism
- Humans
- Inflammation / genetics
- Mice
- Multiple Sclerosis
- Neurons / metabolism
Structure d'affiliation
Groupe de recherche
Financement
- Swiss National Science Foundation - Deciphering transcriptional regulators of T cell fate decisions in CNS autoimmunity [201271]
- Swiss National Science Foundation - Drivers and signatures of neuronal dysfunction in neuroinflammation [185321]
- European Commission - Molecular pathology of anti-viral T cell responses in the central nervous system [865026]
Citation (format ISO)
ROTHAMMER, Nicola et al. G9a dictates neuronal vulnerability to inflammatory stress via transcriptional control of ferroptosis. In: Science advances, 2022, vol. 8, n° 31, p. eabm5500. doi: 10.1126/sciadv.abm5500
Fichiers principaux (1)
Article (Published version)
Fichiers secondaires (1)
Identifiants
- PID : unige:166383
- DOI : 10.1126/sciadv.abm5500
- PMID : 35930635
- PMCID : PMC9355351
ISSN du journal2375-2548