mitoKATP channel activation in the postanoxic developing heart protects E-C coupling via NO-, ROS-, and PKC-dependent pathways
ContributorsSarre, Alexandre; Lange, Norbert; Kucera, Pavel; Raddatz, Eric
Published inAmerican journal of physiology. Heart and circulatory physiology, vol. 288, no. 4, p. H1611-H1619
Publication date2005
Abstract
Keywords
- Animals
- Anoxia/metabolism
- Anti-Arrhythmia Agents/pharmacology
- Atrioventricular Node/physiology
- Chick Embryo
- Chickens
- Decanoic Acids/pharmacology
- Diazoxide/pharmacology
- Enzyme Inhibitors/pharmacology
- Free Radical Scavengers/pharmacology
- Glycine/analogs & derivatives/pharmacology
- Heart/embryology
- Hydroxy Acids/pharmacology
- Membrane Proteins/metabolism
- Myocardial Reperfusion Injury/metabolism
- Myocardium/metabolism
- NG-Nitroarginine Methyl Ester/pharmacology
- Nitric Oxide/metabolism
- Oxidative Stress/drug effects/physiology
- Oxygen/pharmacology
- Potassium Channels
- Protein Kinase C/antagonists & inhibitors/metabolism
- Reactive Oxygen Species/metabolism
- Signal Transduction/physiology
- Sulfhydryl Compounds/pharmacology
- Vasodilator Agents/pharmacology
Affiliation entities
Citation (ISO format)
SARRE, Alexandre et al. mitoKATP channel activation in the postanoxic developing heart protects E-C coupling via NO-, ROS-, and PKC-dependent pathways. In: American journal of physiology. Heart and circulatory physiology, 2005, vol. 288, n° 4, p. H1611–H1619. doi: 10.1152/ajpheart.00942.2004
Main files (1)
Article (Published version)
Identifiers
- PID : unige:81143
- DOI : 10.1152/ajpheart.00942.2004
- PMID : 15550517
Journal ISSN0363-6135