IL-22 capacitates dermal fibroblast responses to TNF in scleroderma

Brembilla, Nicolo; Dufour, Aleksandra Maria; Alvarez, Montserrat; Hugues, Stéphanie; Montanari, Elisa; Truchetet, Marie-Elise; Lonati, Paola; Fontao, Lionel; Gabrielli, Armando; Vettori, Serena; Valentini, Gabriele; Boehncke, Wolf-Henning; Meroni, Pierluigi; Chizzolini, Carlo

doi:10.1136/annrheumdis-2015-207477

Scientific article

English

IL-22 capacitates dermal fibroblast responses to TNF in scleroderma

ContributorsBrembilla, Nicolo; Dufour, Aleksandra Maria; Alvarez, Montserrat; Hugues, Stéphanie; Montanari, Elisa; Truchetet, Marie-Elise; Lonati, Paola; Fontao, Lionel; Gabrielli, Armando; Vettori, Serena; Valentini, Gabriele; Boehncke, Wolf-Henning; Meroni, Pierluigi; Chizzolini, Carlo

Published inAnnals of the rheumatic diseases, vol. 75, no. 9, p. 1697-1705

Publication date2016

Abstract

Interleukin (IL) 22 mRNA in systemic sclerosis (SSc) skin and Th22 cells in SSc peripheral blood are increased, but the role of IL-22 in fibrosis development remains poorly understood.

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Citation (ISO format)

BREMBILLA, Nicolo et al. IL-22 capacitates dermal fibroblast responses to TNF in scleroderma. In: Annals of the rheumatic diseases, 2016, vol. 75, n° 9, p. 1697–1705. doi: 10.1136/annrheumdis-2015-207477

Article (Published version)

Identifiers

PID : unige:79637
DOI : 10.1136/annrheumdis-2015-207477
PMID : 26452537

Journal ISSN0003-4967

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IL-22 capacitates dermal fibroblast responses to TNF in scleroderma

Interleukin (IL) 22 mRNA in systemic sclerosis (SSc) skin and Th22 cells in SSc peripheral blood are increased, but the role of IL-22 in fibrosis development remains poorly understood.

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