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Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

Published inAntioxidants & redox signaling, vol. 23, no. 5, p. 460-489
Publication date2015
Abstract

Phagocytes play a key role in promoting the oxidative stress after ischemic stroke occurrence. The phagocytic NADPH oxidase (NOX) 2 is a membrane-bound enzyme complex involved in the antimicrobial respiratory burst and free radical production in these cells.

Citation (ISO format)
CARBONE, Federico et al. Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2. In: Antioxidants & redox signaling, 2015, vol. 23, n° 5, p. 460–489. doi: 10.1089/ars.2013.5778
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ISSN of the journal1523-0864
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Creation10/26/2015 2:15:00 PM
First validation10/26/2015 2:15:00 PM
Update time03/15/2023 12:03:55 AM
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