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Title

Decreased exhaled nitric oxide may be a marker of cardiopulmonary bypass-induced injury

Authors
Silkoff, P E
Caramori, M
Holtby, H M
Slutsky, A S
Adatia, I
Published in Annals of Thoracic Surgery. 1998, vol. 66, no. 2, p. 532-4
Abstract Nitric oxide is an endothelium-derived vasodilator. Cardiopulmonary bypass may induce transient pulmonary endothelial dysfunction with decreased nitric oxide release that contributes to postoperative pulmonary hypertension and lung injury. Exhaled nitric oxide levels may reflect, in part, endogenous production from the pulmonary vascular endothelium.
Keywords Biological Markers/analysisCardiopulmonary Bypass/adverse effectsChildChild, PreschoolEndothelium, Vascular/metabolismFemaleHeart Defects, Congenital/surgeryHumansInfant, NewbornMaleNitric Oxide/analysis/biosynthesisRespirationTime Factors
Identifiers
PMID: 9725398
Full text
Article (Published version) (335 Kb) - document accessible for UNIGE members only Limited access to UNIGE
Structures
Research group L'hypertension pulmonaire (pédiatrie) (228)
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(ISO format)
BEGHETTI, Maurice et al. Decreased exhaled nitric oxide may be a marker of cardiopulmonary bypass-induced injury. In: Annals of Thoracic Surgery, 1998, vol. 66, n° 2, p. 532-4. https://archive-ouverte.unige.ch/unige:74625

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Deposited on : 2015-08-10

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