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Title

Endotoxin-mediated delayed islet graft function is associated with increased intra-islet cytokine production and islet cell apoptosis

Authors
Molano, R D
Cattan, P
Pileggi, A
Vizzardelli, C
Oliver, R
Ricordi, C
Inverardi, L
Published in Transplantation. 2001, vol. 71, no. 1, p. 125-32
Abstract Primary nonfunction resulting in immediate graft loss is responsible for the failure of a large number of islet transplants. Evidence is accumulating to single out endotoxin contamination of the various reagents needed for islet isolation as a major cause of early graft loss.
Keywords AnimalsApoptosis/drug effectsCells, CulturedCollagenases/pharmacologyCytokines/biosynthesisEndotoxins/pharmacologyIslets of Langerhans/chemistry/cytology/metabolismIslets of Langerhans Transplantation/physiologyMaleMatrix Metalloproteinase 9/pharmacologyMiceMice, Inbred C57BLThermolysin/pharmacology
Identifiers
PMID: 11211177
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Article (Published version) (123 Kb) - document accessible for UNIGE members only Limited access to UNIGE
Structures
Research group La transplantation d'îlots de Langerhans (623)
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BERNEY, Thierry et al. Endotoxin-mediated delayed islet graft function is associated with increased intra-islet cytokine production and islet cell apoptosis. In: Transplantation, 2001, vol. 71, n° 1, p. 125-32. https://archive-ouverte.unige.ch/unige:47294

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Deposited on : 2015-02-26

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