Ischemic preconditioning modulates the expression of several genes, leading to the overproduction of IL-1Ra, iNOS, and Bcl-2 in a human model of liver ischemia-reperfusion
Published inThe FASEB journal, vol. 19, no. 12, p. 1617-1626
Publication date2005
Keywords
- Adult
- Apoptosis
- Biopsy
- Blotting, Western
- Carbohydrates/chemistry
- Caspase 3
- Caspase 8
- Caspase 9
- Caspases/metabolism
- DNA, Complementary/metabolism
- Ephrin-A1/metabolism
- Female
- *Gene Expression Regulation
- Humans
- Immunoblotting
- Immunohistochemistry
- Inflammation
- Interleukin 1 Receptor Antagonist Protein
- *Ischemic Preconditioning
- Liver/metabolism/*pathology
- Male
- Middle Aged
- Nitric Oxide/metabolism
- Nitric Oxide Synthase/metabolism
- Nitric Oxide Synthase Type II/*metabolism
- Oligonucleotide Array Sequence Analysis
- Proto-Oncogene Proteins c-bcl-2/*metabolism
- RNA, Messenger/metabolism
- Reperfusion
- *Reperfusion Injury
- Sialoglycoproteins/*metabolism
- Time Factors
Affiliation entities Not a UNIGE publication
Citation (ISO format)
BARRIER, Alain et al. Ischemic preconditioning modulates the expression of several genes, leading to the overproduction of IL-1Ra, iNOS, and Bcl-2 in a human model of liver ischemia-reperfusion. In: The FASEB journal, 2005, vol. 19, n° 12, p. 1617–1626. doi: 10.1096/fj.04-3445com
Main files (1)
Article (Published version)
Identifiers
- PID : unige:47136
- DOI : 10.1096/fj.04-3445com
- PMID : 16195370
Journal ISSN0892-6638
