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Title

NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes
Authors
Brandt, Karim
Fickentscher, Céline
Boehlen, Françoise
Kruithof, Egbert
De Moerloose, Philippe
Published in Journal of Thrombosis and Haemostasis. 2014, vol. 12, no. 5, p. 779-91
Abstract The antiphospholipid antibody syndrome (APS) is an autoimmune disease associated with arterial or venous thrombosis and/or recurrent fetal loss and is caused by pathogenic antiphospholipid antibodies (aPLA). We recently demonstrated that Toll-like receptor 2 (TLR2) and CD14 contribute to monocyte activation of aPLA.
Identifiers
PMID: 24612386
Full text
Article (Published version) (739 Kb) - document accessible for UNIGE members only Limited access to UNIGE
Structures
Faculté de médecine / Section de médecine clinique / Département de médecine
Research groups Endothélium vasculaire (11)
Anticorps antiphospholipides (12)
Citation
(ISO format) 		BRANDT, Karim et al. NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes. In: Journal of Thrombosis and Haemostasis, 2014, vol. 12, n° 5, p. 779-91. doi: 10.1111/jth.12536 https://archive-ouverte.unige.ch/unige:44083

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Deposited on : 2014-12-22

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