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NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes |
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Authors | ||
Published in | Journal of Thrombosis and Haemostasis. 2014, vol. 12, no. 5, p. 779-91 | |
Abstract | The antiphospholipid antibody syndrome (APS) is an autoimmune disease associated with arterial or venous thrombosis and/or recurrent fetal loss and is caused by pathogenic antiphospholipid antibodies (aPLA). We recently demonstrated that Toll-like receptor 2 (TLR2) and CD14 contribute to monocyte activation of aPLA. | |
Identifiers | DOI: 10.1111/jth.12536 PMID: 24612386 | |
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Research groups | Endothélium vasculaire (11) Anticorps antiphospholipides (12) | |
Citation (ISO format) | BRANDT, Karim et al. NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes. In: Journal of Thrombosis and Haemostasis, 2014, vol. 12, n° 5, p. 779-91. doi: 10.1111/jth.12536 https://archive-ouverte.unige.ch/unige:44083 |