en
Professional article
English

NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes

Published inJournal of thrombosis and haemostasis, vol. 12, no. 5, p. 779-791
Publication date2014
Abstract

The antiphospholipid antibody syndrome (APS) is an autoimmune disease associated with arterial or venous thrombosis and/or recurrent fetal loss and is caused by pathogenic antiphospholipid antibodies (aPLA). We recently demonstrated that Toll-like receptor 2 (TLR2) and CD14 contribute to monocyte activation of aPLA.

Citation (ISO format)
BRANDT, Karim et al. NF-κB is activated from endosomal compartments in antiphospholipid antibodies-treated human monocytes. In: Journal of thrombosis and haemostasis, 2014, vol. 12, n° 5, p. 779–791. doi: 10.1111/jth.12536
Main files (1)
Article (Published version)
accessLevelRestricted
Identifiers
ISSN of the journal1538-7836
471views
0downloads

Technical informations

Creation11/24/2014 3:15:00 PM
First validation11/24/2014 3:15:00 PM
Update time03/14/2023 10:31:48 PM
Status update03/14/2023 10:31:48 PM
Last indexation01/16/2024 3:26:24 PM
All rights reserved by Archive ouverte UNIGE and the University of GenevaunigeBlack