Role of endogenous Fas (CD95/Apo-1) ligand in balloon-induced apoptosis, inflammation, and neointima formation
Publié dansCirculation, vol. 113, no. 15, p. 1879-1887
Date de publication2006
Résumé
Mots-clés
- Animals
- Anti-Inflammatory Agents/metabolism
- Apoptosis
- Carotid Artery Injuries/etiology/pathology
- Catheterization/adverse effects
- Cell Movement
- Cell Proliferation
- Fas Ligand Protein
- Lymphoproliferative Disorders/pathology/physiopathology
- Macrophages/pathology
- Macrophages, Peritoneal/metabolism
- Male
- Membrane Glycoproteins/deficiency/metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Muscle, Smooth, Vascular/pathology
- Myocytes, Smooth Muscle/pathology
- Tumor Necrosis Factors/deficiency/metabolism
- Tunica Intima/pathology
- Tunica Media/pathology
Structure d'affiliation
Groupe de recherche
Citation (format ISO)
MATTER, Christian M et al. Role of endogenous Fas (CD95/Apo-1) ligand in balloon-induced apoptosis, inflammation, and neointima formation. In: Circulation, 2006, vol. 113, n° 15, p. 1879–1887. doi: 10.1161/CIRCULATIONAHA.106.611731
Fichiers principaux (1)
Article (Published version)
Identifiants
- PID : unige:33305
- DOI : 10.1161/CIRCULATIONAHA.106.611731
- PMID : 16606788
ISSN du journal0009-7322