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Role of endogenous Fas (CD95/Apo-1) ligand in balloon-induced apoptosis, inflammation, and neointima formation

Matter, Christian M
Meier, Patricia
von Lukowicz, Tobias
Lohmann, Christine
Schuler, Pia K
Zhang, Dongming
Odermatt, Bernhard
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Published in Circulation. 2006, vol. 113, no. 15, p. 1879-87
Abstract Fas (CD95/Apo-1) ligand (FasL)-induced apoptosis in Fas-bearing cells is critically involved in modulating immune reactions and tissue repair. Apoptosis has also been described after mechanical vascular injury such as percutaneous coronary intervention. However, the relevance of cell death in this context of vascular repair remains unknown.
Keywords AnimalsAnti-Inflammatory Agents/metabolismApoptosisCarotid Artery Injuries/etiology/pathologyCatheterization/adverse effectsCell MovementCell ProliferationFas Ligand ProteinLymphoproliferative Disorders/pathology/physiopathologyMacrophages/pathologyMacrophages, Peritoneal/metabolismMaleMembrane Glycoproteins/deficiency/metabolismMiceMice, Inbred C57BLMice, KnockoutMuscle, Smooth, Vascular/pathologyMyocytes, Smooth Muscle/pathologyTumor Necrosis Factors/deficiency/metabolismTunica Intima/pathologyTunica Media/pathology
PMID: 16606788
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Research group L'athérosclérose (665)
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MATTER, Christian M et al. Role of endogenous Fas (CD95/Apo-1) ligand in balloon-induced apoptosis, inflammation, and neointima formation. In: Circulation, 2006, vol. 113, n° 15, p. 1879-87. doi: 10.1161/CIRCULATIONAHA.106.611731 https://archive-ouverte.unige.ch/unige:33305

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Deposited on : 2014-01-16

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