Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli
Published inEMBO molecular medicine, vol. 2, no. 12, p. 490-503
Publication date2010
Abstract
Keywords
- Animals
- *Apoptosis
- Cell Line
- Cells, Cultured
- Cytochromes c/metabolism
- Female
- GTP Phosphohydrolases/genetics/metabolism
- Humans
- Huntington Disease/genetics/metabolism/*physiopathology
- Male
- Mice
- Microscopy, Electron, Transmission
- Microtubule-Associated Proteins/genetics/metabolism
- Mitochondria/genetics/*physiology/ultrastructure
- Mitochondrial Proteins/genetics/metabolism
- Models, Biological
- Neurons/cytology/metabolism
- Protein Transport
Affiliation entities
Research groups
Citation (ISO format)
COSTA, Veronica et al. Mitochondrial fission and cristae disruption increase the response of cell models of Huntington’s disease to apoptotic stimuli. In: EMBO molecular medicine, 2010, vol. 2, n° 12, p. 490–503. doi: 10.1002/emmm.201000102
Main files (1)
Article
Identifiers
- PID : unige:20858
- DOI : 10.1002/emmm.201000102
- PMID : 21069748
Additional URL for this publicationhttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC3044888/pdf/emmm0002-0490.pdf
Journal ISSN1757-4676