<i>CDKN2A</i> deletion in supratentorial ependymoma with <i>RELA</i> alteration indicates a dismal prognosis: a retrospective analysis of the HIT ependymoma trial cohort

Jünger, Stephanie T; Andreiuolo, Felipe; Mynarek, Martin; Wohlers, Inken; Rahmann, Sven; Klein-Hitpass, Ludger; Dörner, Evelyn; Zur Mühlen, Anja; Velez-Char, Natalia; von Hoff, Katja; Warmuth-Metz, Monika; Kortmann, Rolf-Dieter; Timmermann, Beate; Von Bueren, André; Rutkowski, Stefan; Pietsch, Torsten

doi:10.1007/s00401-020-02169-z

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Title		CDKN2A deletion in supratentorial ependymoma with RELA alteration indicates a dismal prognosis: a retrospective analysis of the HIT ependymoma trial cohort
Authors		Jünger, Stephanie T Andreiuolo, Felipe Mynarek, Martin Wohlers, Inken Rahmann, Sven Klein-Hitpass, Ludger Dörner, Evelyn Zur Mühlen, Anja Velez-Char, Natalia von Hoff, Katja Warmuth-Metz, Monika Kortmann, Rolf-Dieter Timmermann, Beate Von Bueren, André Rutkowski, Stefan Pietsch, Torsten show all authors [1 - 16]
Published in		Acta neuropathologica. 2020, vol. 140, no. 3, p. 405-407
Keywords		Cohort Studies — Cyclin-Dependent Kinase Inhibitor p16 / deficiency — Cyclin-Dependent Kinase Inhibitor p16 / metabolism — Ependymoma / diagnosis — Ependymoma / drug therapy — Ependymoma / metabolism — Ependymoma / pathology — Humans — Prognosis — Supratentorial Neoplasms / metabolism — Supratentorial Neoplasms / pathology — Transcription Factor RelA / metabolism
Identifiers		DOI: 10.1007/s00401-020-02169-z PMID: 32514758 PMCID: PMC7423858
Full text		Article (Published version) (4.4 MB) - Free access
Structures		Faculté de médecine / Section de médecine clinique / Département de pédiatrie, gynécologie et obstétrique
Research group		Oncologie et hématologie pédiatriques (907)
Citation (ISO format)		JÜNGER, Stephanie T et al. CDKN2A deletion in supratentorial ependymoma with RELA alteration indicates a dismal prognosis: a retrospective analysis of the HIT ependymoma trial cohort. In: Acta neuropathologica, 2020, vol. 140, n° 3, p. 405-407. doi: 10.1007/s00401-020-02169-z https://archive-ouverte.unige.ch/unige:157308