Dysfunctional keratinocytes increase dermal inflammation in systemic sclerosis. Results from tissue-engineered scleroderma epidermis

Russo, Barbara; Borowczyk, Julia; Boehncke, Wolf-Henning; Truchetet, Marie-Elise; Modarressi Ghavami, Seyed Ali; Brembilla, Nicolo; Chizzolini, Carlo

doi:10.1002/art.41659

Scientific article

English

Dysfunctional keratinocytes increase dermal inflammation in systemic sclerosis. Results from tissue-engineered scleroderma epidermis

ContributorsRusso, Barbara; Borowczyk, Julia; Boehncke, Wolf-Henning; Truchetet, Marie-Elise; Modarressi Ghavami, Seyed Ali; Brembilla, Nicolo; Chizzolini, Carlo

Published inArthritis and Rheumatology

Publication date2021

Abstract

In systemic sclerosis (SSc) evidence suggests abnormal keratinocyte-fibroblast interactions. We investigated the potential epidermal dysfunction in SSc and its effects on dermal homeostasis.

Affiliation entities

Funding

Citation (ISO format)

RUSSO, Barbara et al. Dysfunctional keratinocytes increase dermal inflammation in systemic sclerosis. Results from tissue-engineered scleroderma epidermis. In: Arthritis and Rheumatology, 2021. doi: 10.1002/art.41659

Article (Accepted version)

Identifiers

PID : unige:148710
DOI : 10.1002/art.41659
PMID : 33497035

Additional URL for this publicationhttps://onlinelibrary.wiley.com/doi/10.1002/art.41659

Datasets

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE156173

Journal ISSN2326-5191

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Creation02/02/2021 15:39:00

First validation02/02/2021 15:39:00

Update time15/03/2023 23:59:52

Status update15/03/2023 23:59:51

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Dysfunctional keratinocytes increase dermal inflammation in systemic sclerosis. Results from tissue-engineered scleroderma epidermis

In systemic sclerosis (SSc) evidence suggests abnormal keratinocyte-fibroblast interactions. We investigated the potential epidermal dysfunction in SSc and its effects on dermal homeostasis.

Technical informations