Impact of Fetuin-A on progression of calcific aortic valve stenosis - The COFRASA - GENERAC study

Kubota, Naozumi; Testuz, Ariane Marie; Boutten, Anne; Robert, Tiphaine; Codogno, Isabelle; Duval, Xavier; Tubiana, Sarah; Hekimian, Guillaume; Arangalage, Dimitri; Cimadevilla, Claire; Kerneis, Caroline; Vahanian, Alec; Messika-Zeitoun, David

doi:10.1016/j.ijcard.2018.03.070

Scientific article

English

Impact of Fetuin-A on progression of calcific aortic valve stenosis - The COFRASA - GENERAC study

ContributorsKubota, Naozumi; Testuz, Ariane Marie; Boutten, Anne; Robert, Tiphaine; Codogno, Isabelle; Duval, Xavier; Tubiana, Sarah; Hekimian, Guillaume; Arangalage, Dimitri; Cimadevilla, Claire; Kerneis, Caroline; Vahanian, Alec; Messika-Zeitoun, David

Published inInternational Journal of Cardiology, vol. 265, p. 52-57

Publication date2018

Abstract

Aortic stenosis (AS) is an active disease, but the determinants of AS progression remain largely unknown. Low levels of Fetuin-A, a powerful inhibitor of ectopic calcification, have been linked to ectopic calcium tissue deposition but its role in AS progression has not been clearly evaluated.

Affiliation entities

Faculté de médecine / Section de médecine clinique / Département de médecine

Research groups

Athérosclérose (591)

Citation (ISO format)

KUBOTA, Naozumi et al. Impact of Fetuin-A on progression of calcific aortic valve stenosis - The COFRASA - GENERAC study. In: International Journal of Cardiology, 2018, vol. 265, p. 52–57. doi: 10.1016/j.ijcard.2018.03.070

Article (Published version)

Identifiers

PID : unige:115823
DOI : 10.1016/j.ijcard.2018.03.070
PMID : 29885700

Journal ISSN0167-5273

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Creation16/11/2018 13:54:00

First validation16/11/2018 13:54:00

Update21/01/2025 16:02:11

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Impact of Fetuin-A on progression of calcific aortic valve stenosis - The COFRASA - GENERAC study

Aortic stenosis (AS) is an active disease, but the determinants of AS progression remain largely unknown. Low levels of Fetuin-A, a powerful inhibitor of ectopic calcification, have been linked to ectopic calcium tissue deposition but its role in AS progression has not been clearly evaluated.

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