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Title

2-Arachidonoylglycerol mobilizes myeloid cells and worsens heart function after acute myocardial infarction

Authors
Schloss, Maximilian J
Horckmans, Michael
Guillamat-Prats, Raquel
Hering, Daniel
Weber, Christian
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Published in Cardiovascular Research. 2019, vol. 115, no. 3, p. 602-613
Abstract Myocardial infarction (MI) leads to an enhanced release of endocannabinoids and a massive accumulation of neutrophils and monocytes within the ischaemic myocardium. These myeloid cells originate from haematopoietic precursors in the bone marrow and are rapidly mobilized in response to MI. We aimed to determine whether endocannabinoid signalling is involved in myeloid cell mobilization and cardiac recruitment after ischaemia onset.
Keywords Myocardial infarctionMonocytesNeutrophilsMonoacylglycerol lipaseCB2 cannabinoid receptor
Identifiers
PMID: 30295758
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Article (Author postprint) (983 Kb) - document accessible for UNIGE members only Limited access to UNIGE (until 2020-02-25)
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Research group Toxicologie analytique et forensique (120)
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SCHLOSS, Maximilian J et al. 2-Arachidonoylglycerol mobilizes myeloid cells and worsens heart function after acute myocardial infarction. In: Cardiovascular Research, 2019, vol. 115, n° 3, p. 602-613. https://archive-ouverte.unige.ch/unige:114680

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Deposited on : 2019-03-01

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