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Interleukin 23-Helper T Cell 17 Axis as a Treatment Target for Pityriasis Rubra Pilaris

Feldmeyer, Laurence
Mylonas, Alessio
Demaria, Olivier
Mennella, Anna
Yawalkar, Nikhil
Hohl, Daniel
Gilliet, Michel
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Published in JAMA Dermatology. 2017, vol. 153, no. 4, p. 304-308
Abstract Treatment of pityriasis rubra pilaris (PRP) is solely based on its resemblance to psoriasis rather than any knowledge of its pathomechanism. Insight into pathogenic mediators of inflammation is essential for targeted and valid treatment options that could replace previous serendipitous therapeutic approaches in refractory PRP.
Keywords AdultBiopsyCytokines/geneticsDermatologic Agents/pharmacology/therapeutic useFollow-Up StudiesHumansInterleukin-23/geneticsMalePityriasis Rubra Pilaris/drug therapy/genetics/pathologyPsoriasis/pathologyRNAMessenger/metabolismTh17 Cells/metabolismTime FactorsTreatment OutcomeUp-RegulationUstekinumab/pharmacology/therapeutic use
PMID: 28122069
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Research group Mécanismes des pathologies de l'inflammation cutanée (919)
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FELDMEYER, Laurence et al. Interleukin 23-Helper T Cell 17 Axis as a Treatment Target for Pityriasis Rubra Pilaris. In: JAMA Dermatology, 2017, vol. 153, n° 4, p. 304-308. doi: 10.1001/jamadermatol.2016.5384 https://archive-ouverte.unige.ch/unige:106970

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Deposited on : 2018-08-10

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