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Title | Published in | Access level | OA Policy | Year | Views | Downloads | |
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Chromatin regulators in the TBX1 network confer risk for conotruncal heart defects in 22q11.2DS | npj genomic medicine | 2023 | 76 | 10 | |||
La maturation cérébrale chez les jeunes et la transition des patients consultants en pédopsychiatrie: y-a-t-il une incohérence? | Cahiers de psychologie clinique | 2021 | 209 | 270 | |||
Using common genetic variation to examine phenotypic expression and risk prediction in 22q11.2 deletion syndrome | Nature Medicine | 2020 | 293 | 0 | |||
Complete Sequence of the 22q11.2 Allele in 1,053 Subjects with 22q11.2 Deletion Syndrome Reveals Modifiers of Conotruncal Heart Defects | American Journal of Human Genetics | 2020 | 356 | 0 | |||
Associations between schizotypal personality features, mentalizing difficulties and thought problems in a sample of community adolescents | Early Intervention in Psychiatry | 2020 | 337 | 181 | |||
Favorable effects of omega-3 polyunsaturated fatty acids in attentional control and conversion rate to psychosis in 22q11.2 deletion syndrome | Neuropharmacology | 2020 | 447 | 3 | |||
Pituitary dysmaturation affects psychopathology and neurodevelopment in 22q11.2 Deletion Syndrome | Psychoneuroendocrinology | 2020 | 372 | 467 | |||
Genetic contributors to risk of schizophrenia in the presence of a 22q11.2 deletion | Molecular Psychiatry | 2020 | 358 | 3 | |||
22q11 microdeletion syndrome and ultra-high risk for psychosis: The role of neurological soft signs as an independent marker of vulnerability for psychosis | Early Intervention in Psychiatry | 2019 | 300 | 1 | |||
Antipsychotics do not influence neurological soft signs in children and adolescents at ultra-high risk for psychosis: a pilot study | Journal of psychiatric practice | 2019 | 274 | 1 | |||
Prevalence of Non-Affective Psychoses in Individuals with Autism Spectrum Disorders: A Systematic Review | Journal of Clinical Medicine | 2019 | 296 | 156 | |||
Attention deficit hyperactivity disorder symptoms as antecedents of later psychotic outcomes in 22q11.2 deletion syndrome | Schizophrenia Research | 2019 | 330 | 121 | |||
Prevalence, course and psychosis-predictive value of negative symptoms in 22q11.2 deletion syndrome | Schizophrenia Research | 2019 | 483 | 270 | |||
Individualized prediction of transition to psychosis in 1,676 individuals at clinical high risk: development and validation of a multivariable prediction model based on individual patient data meta-analysis | Frontiers in Psychiatry | 2019 | 276 | 169 | |||
A Mentalization-Informed Staging Approach to Clinical High Risk for Psychosis | Frontiers in Psychiatry | 2019 | 342 | 181 | |||
Prevalence and Clinical Significance of Symptoms at Ultra High Risk for Psychosis in Children and Adolescents with Obsessive⁻Compulsive Disorder: Is There an Association with Global, Role, and Social Functioning? | Brain Sciences | 2018 | 566 | 225 | |||
Coping Strategies Mediate the Effect of Stressful Life Events on Schizotypal Traits and Psychotic Symptoms in 22q11.2 Deletion Syndrome | Schizophrenia Bulletin | 2018 | 529 | 5 | |||
Variance of IQ is partially dependent on deletion type among 1,427 22q11.2 deletion syndrome subjects | American Journal of Medical Genetics. A | 2018 | 359 | 121 | |||
Cortical morphology development in patients with 22q11.2 deletion syndrome at ultra-high risk of psychosis | Psychological Medicine | 2018 | 555 | 152 | |||
Understanding the pediatric psychiatric phenotype of 22q11.2 deletion syndrome | American Journal of Medical Genetics. A | 2018 | 440 | 1 | |||
Variations in Dysbindin-1 are associated with cognitive response to antipsychotic drug treatment | Nature Communications | 2018 | 471 | 168 | |||
Adolescence is the starting point of sex-dichotomous COMT genetic effects | Translational Psychiatry | 2017 | 429 | 194 | |||
Clinical presentation of Attenuated Psychosis Syndrome in children and adolescents: Is there an age effect? | Psychiatry Research | 2017 | 405 | 2 | |||
No evidence for the presence of genetic variants predisposing to psychotic disorders on the non-deleted 22q11.2 allele of VCFS patients | Translational Psychiatry | 2017 | 568 | 196 | |||
Détection et traitement précoce des sujets à haut risque clinique depsychose : définitions et recommandations | L'encéphale | 2017 | 621 | 13 | |||
The contribution of a neurodevelopment and genetic perspective to our understanding of the pathogenesis of psychotic disorders: the case of 22q11ds and of youth populations | 2017 | 443 | 7 | ||||
Subthreshold Psychosis in 22q11.2 Deletion Syndrome: Multisite Naturalistic Study | Schizophrenia bulletin | 2017 | 506 | 0 | |||
No age effect in the prevalence and clinical significance of ultra-high risk symptoms and criteria for psychosis in 22q11 deletion syndrome: Confirmation of the genetically driven risk for psychosis? | PLOS ONE | 2017 | 505 | 212 | |||
Ultra high risk status and transition to psychosis in 22q11.2 deletion syndrome | World Psychiatry | 2016 | 459 | 0 | |||
Cognitive Decline Preceding the Onset of Psychosis in Patients With 22q11.2 Deletion Syndrome | JAMA psychiatry | 2015 | 648 | 1,118 | |||
Psychiatric Disorders From Childhood to Adulthood in 22q11.2 Deletion Syndrome: Results From the International Consortium on Brain and Behavior in 22q11.2 Deletion Syndrome | The American journal of psychiatry | 2014 | 935 | 8 | |||
Enhanced maternal origin of the 22q11.2 deletion in velocardiofacial and DiGeorge syndromes | American journal of human genetics | 2013 | 643 | 3 |