Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

Carbone, Federico; Teixeira, Priscila Camillo; Braunersreuther, Vincent; Mach, François; Vuilleumier, Nicolas; Montecucco, Fabrizio

doi:10.1089/ars.2013.5778

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Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

Contributeurs/tricesCarbone, Federico; Teixeira, Priscila Camillo; Braunersreuther, Vincent; Mach, François; Vuilleumier, Nicolas; Montecucco, Fabrizio

Publié dansAntioxidants & redox signaling, vol. 23, no. 5, p. 460-489

Date de publication2015

Résumé

Phagocytes play a key role in promoting the oxidative stress after ischemic stroke occurrence. The phagocytic NADPH oxidase (NOX) 2 is a membrane-bound enzyme complex involved in the antimicrobial respiratory burst and free radical production in these cells.

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Citation (format ISO)

CARBONE, Federico et al. Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2. In: Antioxidants & redox signaling, 2015, vol. 23, n° 5, p. 460–489. doi: 10.1089/ars.2013.5778

Article (Published version)

Identifiants

PID : unige:79575
DOI : 10.1089/ars.2013.5778

ISSN du journal1523-0864

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Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

Phagocytes play a key role in promoting the oxidative stress after ischemic stroke occurrence. The phagocytic NADPH oxidase (NOX) 2 is a membrane-bound enzyme complex involved in the antimicrobial respiratory burst and free radical production in these cells.

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